Heart Condition Case Study

Heart Condition Case Study Samantha O’Shea Course: Healthcare Support Module: Human Growth and Development Introduction I got permission from Ned to do my assignment on him. Some of his information has been changed for confidentiality reasons. Ned is sixty and the oldest of nine children. Ned has heart condition which is hereditary in his family. His mother had an enlarged heart which the never knew till after she died. Both his father and younger sister also have a heart condition. His father died from the heart condition over twenty years ago. She died at the age of forty two years of age, five years after giving birth to Ned’s younger sister. She also had eight of her children at home which included a set of twins. Ned had a heart attack about ten years ago while he was in. He found it hard to breath and pains in his chest but didn’t realise he was taking the heart attack. When he was brought to the hospital there was test done and the discovered that he had suffered a heart attack. Ned had a second one year’s later. Before Ned had the heart attack he an unhealthy diet and a heavy smoker. He worked full time in a hotel but now is only working part time. Ned enjoys working as he gets to meet other people and it keeps him busy. A heart attack is when arteries become blocked or damaged. Plaque builds up in the arteries causing a blood clot. The muscles become damaged or die which cause the heart attack. Symptoms are: Chest pain which is most common. Shortness of breath Upper body pain i.e. jaw, back and arms Weakness Tiredness To diagnose a heart attack is getting an Electrocardiogram (ECG). Can be done in a hospital or you’re local GPs. (Irish Heart Foundation 2015) Physical, Intellectual, Emotional and Social (P.I.E.S) of development during late adulthood Late adulthood is from the age of sixty-five plus. Their development is completed. Physical People in this age group will be going through different physical development caused by aging, their walking be getting slower. In some cases people will get lose some of their fine motor skills such as writing or open a door. Also some people’s ears grow. By 65 a lot of people will have grey hair, wrinkles, their muscles weakening and skin will losing or not as flexible. Also their bones will get weaker. According to Erikson’s, he believes that people in the 65+ age have more health problems when they get older. Examples off health problems such as diabetes, refluxes, heart condition dementia and motor neuron (MN). Intellectual A lot of people will be fully developed. In some cases some people will lose their memory from dementia or their thinking will start to slow down. Some people won’t be quick enough to remember stuff e.g. birthdays. People won’t be quick enough to solve problems. According to Cliff Notes older people will learn work or information a bit slower than other people or even to remember any work. Their memory won’t be as good as a younger person. But believe that they can still learn as much as a younger person. (Cliff Notes 2014) Emotional Some people could be upset because they are losing people around them such as friends and family. They could be upset because they have to retire. They could be used to working and they don’t want to be sitting around doing nothing which could cause a person to become depressed or lonely. Some people could be emotional because of an illness they have or they could be dying According to Kubler – Ross a person has to go through the different stages of dying. There are five different stages which are Denial, Anger Bargaining, Depression and Acceptance. A person will go through a lot of different emotions from an illness or dying and will go through Kubler – Ross theory. (Cliff Notes 2014) Social A lot of people at this age will have a lot of people around them such as family and friends. If they are retire the can spend a lot more time with people by doing activities or even some lunch. Another side of retiring people will become anti social because the fell that they have know one around them, which can cause people to become depressed. According to Erikson’s Integrity VS Despair 65 + a lot of older people recall on their past. They look back at their pride and their dignity. Erikson believes that people in this age category accept death. He then believes that elderly people start isolating themselves from peer groups. They start looking back at their achievements, mistakes and/or missed opportunities. He often believes that people at this age will start isolating themselves from others. Physical, Intellectual, Emotional and Social (P.I.E.S) of development of individual as well as your own personal prejudices, fears and anxieties Ned’s Physical, Intellectual, Emotional and Social (P.I.E.S) didn’t change much throughout his late adulthood from the heart attack Physical Before Ned had his heart attack he lived an unhealthy lifestyle which included unsociable working hours and he was a heavy smoker. He only works part time now. After having the heart attack he became more tired and he wouldn’t be able to do much work. Ned does certain jobs and he can become weak or breathless. Intellectual Ned has his full senses in his age. He has great memory and is able to solve problems. Ned is able to interact with other people. Ned’s intellectual development didn’t change. Emotional Ned is nervous because he is retiring in a few years. He is used to working because he is working since he was fourteen or fifteen so he has been working for over fifty years. After Ned having the heart attack he was upset because he was going through a difficult time but now he is fine. Social Ned has a good social life. He sees his family regularly and also his friends. He enjoys going to occasions such as weddings to have a good time. Also he enjoys going to his sibling’s house for dinner and a chat. He also has a good relationship with some of his work mates They all started in one of the factories in town and then got the job in the hotel. My own personal prejudice, fears and anxieties on Ned’s Hearth Condition. I am worried that Ned will take another heart attack that would affect his development. It could affect him physically by getting weaker and he mightn’t be able to return to work. Intellectual it could affect him by him losing his memory which wouldn’t be good for Ned. Emotionally and socially it could affect him by being depressed by not able to work if he has another heart attack. Ned could become anti sociable and depressed. Variations/Factors affecting the individual at this stage of life Ned at this stage is still working part time and is enjoying it as is working since he was young. Ned’s health doesn’t affect him that much, unless he is doing something that is making his heart over work such as lifting something to heavy or rushing. It could make him tired or breathless. If Ned does something to put straining on his heart it could affect him by having another heart attack. A way he could manage it is by not lifting anything to heavy or eating unhealthy food which will help his heart. He could go for little walks to keep fit. Another thing that could affect Ned is if he over does something like too much exercise, pushing something he can’t. Ned would be putting too much strain on his heart which could leave him breathless, tired or even in hospital. Ned could do some light exercise. When Ned hits retirement his routine will be out because he won’t be working. Ned won’t be able to interact with as many people because he won’t be working and also he is a single man. He won’t have anyone to talk to. Another way is that Ned stops or cuts down on smoking as it affects his health. Recommendations to respond confidently to needs of person I would recommend Ned to start getting involved with age action so that he can get involved with other people. He won’t become isolated or non sociable with other people. He can meet up with people for an hour or two a week for a chat and a cup of tea. I also recommend that Ned gets involved with the day care centre the days he isn’t working. He can interact with other people and also get his dinner there for little as five euro. In the day care centre they provide activities. I recommend he joins the Active Retirement Association Tullamore (T.A.R.A). This is on one day a week for people over the age of 55. The get to go on trips over Ireland, have social gatherings, exercise and creative opportunities. Another organisation Ned could join is the Arden View resource centre. It allows the people to meet up and socialise with other people, go on day trips, and get to do activities. Ned could do any of these organisations that won’t be too stressful or to harmful on his heart. Ned will get to interact with other people and become more sociable. He would enjoy the activities and socialising with other people because he likes talking and getting involved. Also these would help Ned to get used to being in organisations because he will be retiring in the next year or two. If he starts now he will get used to being in organisations and where as if he leaves it to the last minute he mightn’t have the confidence to join. Conclusion From doing this assignment I learnt that Ned’s family heart condition is genetic in his family. His family should be getting themselves checked for heart condition because the could be affected. I also learnt more about a heart disease and what it’s physical and mental affects are on someone. I learnt that it could kill of some of your heart and I learnt more on the signs and symptoms such as the upper body pain. Bibliography Book Creative Training Book 2015 Web sites Cliffs Notes (2014) Development in Late Adulthood [online], Available: http://www.cliffsnotes.com/sciences/psychology/psychology/developmental-psychology-age-13-to-65/development-in-late-adulthood [accessed 30th March 2015] Cliffs Notes (2014) Intelligence and Memory age 65 [online], Available: http://www.cliffsnotes.com/sciences/psychology/development-psychology/physical-cognitive-development-65/intelligence-and-memory-age-65 [accessed 30th March 2015] Irish Heart Foundation (2015) Heart Attack [online], Available: https://www.irishheart.ie/iopen24/heart-attack-t-7_19_61.html [accessed 29th March 2015] 1 5M4339Samantha O’Shea

The Nature of Viruses

Viruses are sub-cellular agents of infection that must utilize the cellular machinery of bacteria, plants or animals in order to reproduce. Composed of a single strand of genetic material (DNA or RNA) encased in a protein capsid, a virus is too small to be seen by standard light microscopy; indeed, most are less than one hundredth the size of a bacterium. Specific proteins on the viral capsid attach to receptors on the host cell; this attachment process is essential to viral infectivity and explains why viruses may only infect the cells of certain species or may only infect certain cells or tissues within a given host species. While the infecting virus triggers an immune response in the host, some are capable of suppressing that response by infecting and killing cells that control immunity (e. g. HIV attacks lymphocytes). In addition, while most infected cells are destroyed by viral replication, some viruses enter a latent phase within cells, reactivating in the future to produce chronic or relapsing infections. Many viruses use specific carriers (known as vectors) such as mosquitoes, ticks, bats and rodents that transmit the virus to a susceptible host while others are spread between individuals via blood contact or through respiratory, intestinal or sexual secretions. Of special concern is the fact that mutations within the viral genome may allow viruses to skip from one host (e. g. birds, swine, monkeys) to another (e. g. humans), unleashing pandemics. Many common human infections are produced by viruses; these include the common cold, influenza, mononucleosis, herpes infections (including shingles), viral hepatitis (A, B, C and others), HIV, viral gastroenteritis, conjunctivitis, viral pneumonia, encephalitis, viral meningitis and viral infections of the heart, including pericarditis and myocarditis. While viruses do not respond to antibiotics, specific antiviral agents may control (though not cure) chronic disease (such as HIV, Hepatitis B and Hepatitis C) or may modify the severity of acute infection (as in influenza and herpes infections). However, in most viral infections, treatment is, for now, purely symptomatic and supportive. On the other hand, vaccines are capable of preventing some viral infections (e. g. herpes simplex, measles, mumps, rubella, varicella, Hepatitis B) or reducing the severity of an acute infection (e. g. influenza). Beyond the acute or chronic illness that they produce, some viral infections (such and Hepatitis C and certain strains of herpes simplex) are known to be precursors of malignancy. Finally, many researchers suspect that viruses play a role in the pathogenesis of chronic illnesses such as multiple sclerosis and autoimmune disorders. ttp://naturesblog. blogspot. com/2013/01/the-nature-of-viruses. html The Nature of Viruses Viruses exist in two different states, the extracellular infectious particle or virion and the intracellular state consisting of viral nucleic acid. The capsid may be a polyhedron or a helix, or a combination of both (in some phages). Viruses are infective micro ¬o rganisms that show several differences from typical microbial cells. 1. Size. The size range of viruses is from about 20 to 300 nm. On the whole, viruses are much smaller than bacteria. Most animal viruses and all plant viruses and phages are invisible under the light microscope. 2. Simple structure. Viruses have very simple structures. The simplest viruses are nucleoprotein particles consisting of genetic material (DNA or RNA) surrounded by a protein capsid. In this respect they differ from typical cells which arc made up) of proteins, carbohydrates, lipids and nuc1eicacids. The more complex viruses contain lipids and carbohydrates in addition to proteins and nucleic acids, e. g. the enveloped viruses 3. Absence of cellular structure. Viruses do not have any cytoplasm, and thus cytoplasmic organelles like mitochondria, Golgi complexes, lysosomes, ribosomes, etc. , are absent. They do not have any limiting cell membrane. They utilize the ribosomes of the host cell for protein synthesis during reproduction. 4. No independent metabolism. Viruses cannot multiply outside a living cell. No virus has been cultivated in a cell-free medium. Viruses do not have an independent metabolism. They are metabo ¬lically inactive outside the host cell because they do not posses enzyme systems and protein synthesis machinery. Viral nucleic acid replicates by utilizing the protein synthesis machinery of the host. It codes for the synthesis of a limited number of viral proteins, including the subunits or capsomeres of the capsid, the tail protein and some enzymes concerned Viruses have only one nucleic acid, either DNA or RNA. Typical cells have both DNA and RNA. Genomes of certain with the synthesis or the release of virions. 5. Nucleic acids. RNA viruses can be transcribed into complementary DNA strands in the infected host cells, e. g. Rous Sarcoma Virus (RSV). Such RNA viruses are therefore also called RNA-DNA viruses. 6. Crystallization. Many of the smaller viruses can be crystallized, and thus behave like chemicals. 7. No growth and division. Viruses do not have the power of growth and division. A fully formed virus does not increase in, size by addition of new molecules. The virus itself cannot divide. Only its genetic material (RNA or DNA) is capable of reproduction and that too only in a host cell. It will thus be seen that viruses do not show all the characteris ¬tics of typical living organisms. They, however, possess two funda ¬mental characteristics of living systems. Firstly, they contain nucleic acid as their genetic material. The nucleic acid contains instructions for the structure and function of the virus. Secondly, they can reproduce themselves, even if only by using the host cells synthesis machinery. Viral genomes The nucleic acid comprising the genome may be single-stranded or double-stranded, & in a linear, circular or segmented configuration. Single-stranded virus genomes may be: †¢ positive (+)sense, i. e. of the same polarity (nucleotide sequence) as mRNA †¢ negative (-)sense Ambisense – a mixture of the two. N/B. Virus genomes range in size from approximately 3,200 nucleotides (nt) to approximately 1. 2 million base pairs Unlike the genomes of all cells, which are composed of DNA, virus genomes may contain their genetic information encoded in either DNA or RNA. Since viruses are obligate intracellular parasites only able to replicate inside the appropriate host cells, the genome must contain information encoded in a form which can be recognized & decoded by the particular type of cell parasitized. Thus, the genetic code employed by the virus must match or at least be recognized by the host organism. Similarly, the control signals which direct the expression of virus genes must be appropriate to the host. Many of the DNA viruses of eukaryotes closely resemble their host cells in terms of the biology of their genomes: Some DNA virus genomes are complexed with cellular histones to form a chromatin-like structure inside the virus particle. http://expertscolumn. com/content/nature-viruses http://www. mcb. uct. ac. za/tutorial/virorig. html Viral evolution Viral evolution is a subfield of evolutionary biology and virology that is specifically concerned with the evolution of viruses. Many viruses, in particular RNA viruses, have short generation times and relatively high mutation rates (on the order of one point mutation or more per genome per round of replication for RNA viruses). This elevated mutation rate, when combined with natural selection, allows viruses to quickly adapt to changes in their host environment. Viral evolution is an important aspect of the epidemiology of viral diseases such as influenza (influenza virus), AIDS (HIV), and hepatitis (e. . HCV). It also causes problems in the development of successful vaccines and antiviral drugs, as resistant mutations often appear within weeks or months after the beginning of the treatment. One of the main theoretical models to study viral evolution is the quasispecies model, as the viral quasispecies. | Origins Viruses are ancient. Studies at the molecular level have revealed relationships between viruses infecting organisms from each of the three domains of life, and viral proteins that pre-date the divergence of life and thus the last universal common ancestor. 1] This indicates that viruses emerged early in the evolution of life and existed before modern cells. [2] There are three classical hypotheses on the origins of viruses: Viruses may have once been small cells that parasitised larger cells (the degeneracy hypothesis[3][4] or reduction hypothesis[5]); some viruses may have evolved from bits of DNA or RNA that â€Å"escaped† from the genes of a larger organism (the vagrancy hypothesis[6] or escape hypothesis); or viruses could have evolved from complex molecules of protein and nucleic acid at the same time as cells first appeared on earth (the virus-first hypothesis). 5] None of these hypotheses was fully accepted: the regressive hypothesis did not explain why even the smallest of cellular parasites do not resemble viruses in any way. The escape hypothesis did not explain the complex capsids and other structures on virus particles. The virus-first hypothesis was quickly dismissed because it contravened the definition of viruses, in that they require host cells. [5] Virologists are, however, beginning to reconsider and re-evaluate all three hypotheses. [7][8] http://en. wikipedia. org/wiki/Viral_evolution Evolution Time-line of paleoviruses in the human lineage[9] Viruses do not form fossils in the traditional sense, because they are much smaller than the grains of sedimentary rocks that fossilize plants and animals. However, the genomes of many organism contain endogenous viral elements (EVEs). These DNA sequences are the remnants of ancient virus genes and genomes that ancestrally ‘invaded' the host germline. For example, the genomes of most vertebrate species contain hundreds to thousands of sequences derived from ancient retroviruses. These sequences are a valuable source of retrospective evidence about the evolutionary history of viruses, and have given birth to the science of paleovirology. 9] The evolutionary history of viruses can to some extent be inferred from analysis of contemporary viral genomes. The mutation rates for many viruses have been measured, and application of a molecular clock allows dates of divergence to be inferred. [10] Viruses evolve through changes in their DNA (or RNA), some quite rapidly, and the best adapted mutants quickly outnumber their less fit counterparts. In this sense their evolution is Darwinian, just like that of their host organisms. [11] The way viruses reproduce in their host cells makes them particularly susceptible to the genetic changes that help to drive their evolution. 12] The RNA viruses are especially prone to mutations. [13] In host cells there are mechanisms for correcting mistakes when DNA replicates and these kick in whenever cells divide. [13] These important mechanisms prevent potentially lethal mutations from being passed on to offspring. But these mechanisms do not work for RNA and when an RNA virus replicates in its host cell, changes in their genes are occasionally introduced in error, some of which are lethal. One virus part icle can produce millions of progeny viruses in just one cycle of replication, therefore the production of a few â€Å"dud† viruses is not a problem. Most mutations are â€Å"silent† and do not result in any obvious changes to the progeny viruses, but others confer advantages that increase the fitness of the viruses in the environment. These could be changes to the virus particles that disguise them so they are not identified by the cells of the immune system or changes that make antiviral drugs less effective. Both of these changes occur frequently with HIV. [14] Phylogenetic tree showing the relationships of morbilliviruses of different species[15] Many viruses (for example, influenza A virus) can â€Å"shuffle† their genes with other viruses when two similar strains infect the same cell. This phenomenon is called genetic shift, and is often the cause of new and more virulent strains appearing. Other viruses change more slowly as mutations in their genes gradually accumulate over time, a process known as genetic drift. [16] Through these mechanisms new viruses are constantly emerging and present a continuing challenge to attempts to control the diseases they cause. [17][18] Most species of viruses are now known to have common ancestors, and although the â€Å"virus first† hypothesis has yet to gain full acceptance, there is little doubt that the thousands of species of modern viruses have evolved from less numerous ancient ones. 19] The morbilliviruses, for example, are a group of closely related, but distinct viruses that infect a broad range of animals. The group includes measles virus, which infects humans and primates; canine distemper virus, which infects many animals including dogs, cats, bears, weasels and hyaenas; rinderpest, which infects cattle and buffalo; and other viruses of seals, porpoises and dolphins. 20] Although it not possible to prove which of these rapidly evolving viruses is the earliest, for such a closely related group of viruses to be found in such diverse hosts suggests a possible ancient common ancestor. [21] The Nature of Viruses Viruses are sub-cellular agents of infection that must utilize the cellular machinery of bacteria, plants or animals in order to reproduce. Composed of a single strand of genetic material (DNA or RNA) encased in a protein capsid, a virus is too small to be seen by standard light microscopy; indeed, most are less than one hundredth the size of a bacterium. Specific proteins on the viral capsid attach to receptors on the host cell; this attachment process is essential to viral infectivity and explains why viruses may only infect the cells of certain species or may only infect certain cells or tissues within a given host species. While the infecting virus triggers an immune response in the host, some are capable of suppressing that response by infecting and killing cells that control immunity (e. g. HIV attacks lymphocytes). In addition, while most infected cells are destroyed by viral replication, some viruses enter a latent phase within cells, reactivating in the future to produce chronic or relapsing infections. Many viruses use specific carriers (known as vectors) such as mosquitoes, ticks, bats and rodents that transmit the virus to a susceptible host while others are spread between individuals via blood contact or through respiratory, intestinal or sexual secretions. Of special concern is the fact that mutations within the viral genome may allow viruses to skip from one host (e. g. birds, swine, monkeys) to another (e. g. humans), unleashing pandemics. Many common human infections are produced by viruses; these include the common cold, influenza, mononucleosis, herpes infections (including shingles), viral hepatitis (A, B, C and others), HIV, viral gastroenteritis, conjunctivitis, viral pneumonia, encephalitis, viral meningitis and viral infections of the heart, including pericarditis and myocarditis. While viruses do not respond to antibiotics, specific antiviral agents may control (though not cure) chronic disease (such as HIV, Hepatitis B and Hepatitis C) or may modify the severity of acute infection (as in influenza and herpes infections). However, in most viral infections, treatment is, for now, purely symptomatic and supportive. On the other hand, vaccines are capable of preventing some viral infections (e. g. erpes simplex, measles, mumps, rubella, varicella, Hepatitis B) or reducing the severity of an acute infection (e. g. influenza). Beyond the acute or chronic illness that they produce, some viral infections (such and Hepatitis C and certain strains of herpes simplex) are known to be precursors of malignancy. Finally, many researchers suspect that viruses play a role in the pathogenesis of chronic illnesses such as multiple sclerosis and autoimmune disorders. development of viruses (images)

Watson and Rayners Unconditional Stimulus Experiment on...

Watson and Rayner set out to substantiate his theory by recruiting a subject, who basically stayed in a hospital attached John Hopkins University, and the conducting an experiment on the chosen infant. Their aim was to classically condition the infant to fear a white laboratory rat. The infant, Albert B. or â€Å"Little Albert†, was a physically healthy and emotionally stable 9-month old boy (Watson Rayner, 1920, p. 1). He was described as a very relaxed baby who hardly ever cried. In the emotional tests, before conditioning, Albert was exposed to a series of objects, which included fire, cotton wool, a monkey, a dog, a white rabbit, a mask with hair, and a white laboratory rat. He had not shown a scared or nervous response toward these neutral stimuli, and particularly adored the white laboratory rat. (chaopret 7, p. 239). Firstly, the researchers attempted to identify an unconditioned stimulus. They carried this out by making a loud noise, hitting a hammer against a steel bar, and observing whether the infant responds in fear (learning+c, p.74). In this case, Albert started to cry, which suggested that he was afraid. This gave them the method of testing several important aspects of the experiment. The actual practise of conditioning only started when Albert was 11 months old. The first step Watson and Raymer took towards conditioning Little Albert, was paring the loud noise, unconditioned stimulus, with the white rat, conditioned stimulus. The responses were fear of theShow MoreRelatedInvestigating Crime and Relevant Issues Essay7311 Words   |  30 Pagescontribution to establishing the study of criminal behaviour and was responsible for moving the study of criminology towards scientific research, indicating that biological influences could affect criminality. However Lombroso’s experiments cannot be considered to be valid because he never compared his criminal sample to a non-criminal control group or took account of racial and ethical differences. He recorded inaccurate measurements, which were not based on proper